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The Pharmaceutical Society of Australia (PSA) is the peak national professional pharmacy organisation, representing Australia's 32,000 pharmacists in all sectors and across all locations, working in or towards a career in pharmacy. Registered training organisation code: 122206 ABN:49 008 532 072 ACN:008 532 072
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Insulin resistance and hyperandrogenism are the leading causes of polycystic ovary syndrome (PCOS). Therefore, it has great significance to study the expression levels of PSA, nesfatin-1, and AMH. To provide some reference for clinical diagnosis and treatment of polycystic ovary syndrome (PCOS), the expression levels of PSA, nesfatin-1, and AMH in serum of patients with polycystic ovary syndrome (PCOS) were investigated. The experimental group consisted of 200 patients with polycystic ovary syndrome treated in Shanghai Huashan Hospital from July 2018 to July 2019. The control group consisted of 150 healthy women without pregnancy. The PSA, nesfatin-1, and AMH levels in serum were detected by chemiluminescence immunoassay (CLIA) and enzyme-linked immunosorbent assay (ELISA). The serum levels of prostate-specific antigen (PSA) and anti-Mullerian hormone (AMH) were 16.53 0.67pg/ml and 10.75 4.02pg/ml in the experimental group (PCOS patients), which were significantly higher than those in the control group (3.27 0.43pg/ml and 5.18 1.84pg/ml, respectively), while the inhibitive factors in the experimental group (1.89 0.99mg/ml) were significantly higher than those in the control group (1.10 0.97mg/ml). There was no significant difference in nesfatin-1. The levels of PSA and nesfatin-1, nesfatin-1, and AMH and the levels of PSA and AMH in patients with polycystic ovary syndrome were positively correlated, and the differences were statistically significant. The levels of PSA, nesfatin-1, and AMH in patients with polycystic ovary syndrome of different ages were different, and the differences were significant and negatively correlated with the age increasing. PSA, nesfatin-1, and AMH levels in patients with polycystic ovary syndrome were significantly different from those in control nonpregnant women. There was a certain correlation between the levels of PSA, nesfatin-1, and AMH, and age. The results have specific clinical reference significance for the diagnosis and treatment of patients with polycystic ovary syndrome.
2. NHS UKMi 2020, Medicines Q&As. What are clinically significant drug interactionswith tobacco smoking?, viewed 28 October 2020. At -content/uploads/2020/03/UKMi_QA_Interactions-with-tobacco_update_Jul-2020.pdf
11. Hiemke C et al, 2018. Consensus Guidelines for Therapeutic DrugMonitoring in Neuropsychopharmacology: Update 2017, Pharmacopsychiatry;51:9-62.At -connect.com/products/ejournals/pdf/10.1055/s-0043-116492.pdf
Specimens should not be drawn immediately after digital rectal examination (DRE), prostatic massage, or transrectal ultrasound (TRUS). PSA sampling should not be performed for at least six weeks after prostatic biopsy.
Values obtained with different assay methods should not be used interchangeably in serial testing. It is recommended that only one assay method be used consistently to monitor each patient's course of therapy. This procedure does not provide serial monitoring. It is intended for one-time use only. If serial monitoring is required, please use the serial monitoring number 480640 to order.
The interpretive guidelines provided for percent free PSA are based on a population of men with normal digital rectal exam (DRE) and total PSA between 4.0 and 10.0 ng/mL. Catalona and coworkers2 did not make specific recommendations regarding the use of percent free PSA for any other population of men.
Patients taking finasteride, an alpha-reductase inhibitor, will have diminished levels of PSA. PSA complexes are more stable than free PSA.1 The serum levels of both total and free PSA increase with prostate manipulation but the free returns to premanipulation concentrations quicker. This can result in a transient elevation in percent free PSA.
In general, serum PSA levels increase due to physical changes to prostate architecture caused by trauma, infection, inflammation, prostate manipulation, benign prostatic hypertrophy (BPH), or malignancy.3,4 The sensitivity of PSA levels to these changes serves as the basis for the clinical use of the test. The PSA concentration in the serum of healthy men is a millionfold lower than that in seminal fluid. PSA in seminal fluid is predominantly free or uncomplexed. In serum, the majority of PSA is bound to inhibitors, including α1-antichymotrypsin (ACT) and α2-macroglobulin (A2M). Measured total PSA consists of free and ACT-bound, since PSA complexed to A2M is not immunologically detectable.
Alternatively, percent free PSA may be used to determine the relative risk of prostate cancer in individual men.2 The following table lists the probability of prostate cancer for men with nonsuspicious DRE results and total PSA between 4.0 and 10.0 ng/mL, by patient age (see Limitations). See table.
The LOINC codes are copyright 1994-2021, Regenstrief Institute, Inc. and the Logical Observation Identifiers Names and Codes (LOINC) Committee. Permission is granted in perpetuity, without payment of license fees or royalties, to use, copy, or distribute the LOINC codes for any commercial or non-commercial purpose, subject to the terms under the license agreement found at Additional information regarding LOINC codes can be found at LOINC.org, including the LOINC Manual, which can be downloaded at LOINC.org/downloads/files/LOINCManual.pdf
Free Prostate Specific Antigen (Free PSA) is one of two forms of PSA occuring in human serum. The other is complexed or Total PSA. Free PSA is primarily ordered when a patient has a moderately elevated total PSA that does not appear to be caused by a non-cancer-related condition. The results give the doctor additional information about whether a patient is at an increased risk of having prostate cancer and help with the decision of whether to biopsy the prostate.
We are very excited to be launching the Hedgeye REITs research vertical, and hope you can join us on Monday, April 5th at 10am ET for the inaugural call and presentation. We will open the call by briefly reviewing our background and experience, research process and the REIT universe, and then spend the bulk of the call presenting two best ideas in the self storage subsector: New Best Idea Short Extra Space Storage (EXR) as a hedge / financing vehicle against a New Best Idea Long Public Storage (PSA). Specifically, we will discuss why the long and contrarian short can generate a 30% IRR over the next 12-18 months, which is a homerun in the relatively low-octane REIT space.
In the coming weeks there will be a follow-up call to discuss forthcoming best ideas in the apartments and single-family rental REIT subsectors. The initial launch will focus on the shorter-duration REITs, with longer duration subsectors to be rolled out later in the 2Q / 3Q.
Self-storage has been a relative outperformer among REITs during the pandemic, with best-of-breed Extra Space (EXR) and other operators breaking the traditional seasonal pattern and maintaining high occupancy rates through the weaker fall and winter leasing seasons. This has set up extremely tough Y/Y occupancy comps in 2H21, the toughest comps EXR has seen in at least 10 years. A return to seasonal patterns in the back half of 2021 will likely result in several hundred basis points of occupancy loss, set against guidance calling for 4.25% to 5.50% same store revenue growth in 2021. Growth will likely decelerate for several quarters heading into 2022, and the second derivative on both NOI and FFO growth will be negative in 2022. Risk is skewed to the downside, and we will discuss how we view a short of EXR as a way to finance a long of PSA and enhance returns.
Low-risk patients (defined by using commonly accepted categories such as American Urological Association guidelines) are unlikely to have disease identified by bone scan. Accordingly, bone scans are generally unnecessary in patients with newly diagnosed prostate cancer who have a PSA 041b061a72